Research Article

Protein Pump Inhibitors Esomeprazole and Pantoprazole Increase the Chemosensitivity of CML cells against Imatinib

Volume: 40 Number: 4 December 29, 2018
EN

Protein Pump Inhibitors Esomeprazole and Pantoprazole Increase the Chemosensitivity of CML cells against Imatinib

Abstract

Introduction: Proton pump inhibitors (PPIs) largely used drug to treat gastroesophageal disease such as gastric ulcers. Moreover, in recent years, several studies suggest that PPIs have important anti-cancer effect in monotherapy and or combination with chemotherapy.

Objective: The aim of this study was to investigate whether esomeprazole and pantoprazole exhibit anti-cancer effect alone or could enhance chemosensitivity of CML cell line K562 to imatinib.

Method: Human chronic myeloid leukemia (CML) cells were cultured and treated with different concentrations of esomeprazole, pantoprazole, and imatinib alone. Also these cells exposed to imatinib + esomeprazole and imatinib + pantoprazole combinations, respectively and incubated 24 h. The antiproliferative activities of the (PPIs) alone or in combination of imatinib was evaluated using the XTT colorimetric assay.

Results: According to experimental data, neither PPIs showed any cytotoxic­ity on the K562 cell line at all concentrations except at 500 and 1000 µM. However, when combined with imatinib separately, they were found to have significant anti-cancer effects on K562 cells when compared to cell lines treated with imatinib alone (p<0.05).

Conclusions: Taken together, the inhibition of V-ATPase via esomeprazole and pantoprazole might enhance the chemosensitivity of imatinib in CML cells. However, further studies are needed to be able to utilize PPIs in CML.

Keywords

References

  1. 1. Ergül M, Ergül M, Tutar Y. Important Anti-Cancer Applications of Protein Based Nanoparticles. Current Proteomics 2013;10:334-340.
  2. 2. Inoue A, Kobayashi CI, Shinohara H, et al. Chronic myeloid leukemia stem cells and molecular target therapies for overcoming resistance and disease persistence. Int J Hematol 2018;108(4):365-370.
  3. 3. Dalgıç CT, Kaymaz BT, Özkan MC, et al. Investigating the Role of JAK/STAT Pathway on Dasatinib-Induced Apoptosis for CML Cell Model K562. Clin Lymphoma Myeloma Leuk 2015;15.
  4. 4. Li X, Pang J, Xue W, et al. Inducible SHP-2 activation confers resistance to imatinib in drug-tolerant chronic myeloid leukemia cells. Toxicol Appl Pharmacol 2018;360:249-256.
  5. 5. Udelnow A, Kreyes A, Ellinger S, et al. Omeprazole inhibits proliferation and modulates autophagy in pancreatic cancer cells. PLoS One 2011;6(5):e20143.
  6. 6. Lu ZN, Tian B, Guo XL. Repositioning of proton pump inhibitors in cancer therapy. Cancer Chemother Pharmacol 2017;80(5):925-937.
  7. 7. Wang X, Liu C, Wang J, et al. Proton pump inhibitors increase the chemosensitivity of patients with advanced colorectal cancer. Oncotarget 2017;8(35):58801-58808.
  8. 8. Lindner K, Borchardt C, Schöpp M, et al. Proton pump inhibitors (PPIs) impact on tumour cell survival, metastatic potential and chemotherapy resistance, and affect expression of resistance-relevant miRNAs in esophageal cancer. J ExpClin Cancer Res 2014;33:73.

Details

Primary Language

English

Subjects

Health Care Administration

Journal Section

Research Article

Publication Date

December 29, 2018

Submission Date

December 19, 2018

Acceptance Date

December 25, 2018

Published in Issue

Year 1970 Volume: 40 Number: 4

AMA
1.Ergül M, Ergül M. Protein Pump Inhibitors Esomeprazole and Pantoprazole Increase the Chemosensitivity of CML cells against Imatinib. CMJ. 2018;40(4):351-355. doi:10.7197/223.vi.499367

Cited By