Araştırma Makalesi

Three case reports of alkaptonuria, a rare metabolic disease, in two first-degree relatives and one patient

Cilt: 32 Sayı: 2 11 Haziran 2010
PDF İndir
EN TR

Three case reports of alkaptonuria, a rare metabolic disease, in two first-degree relatives and one patient

Abstract

Alkaptonuria is a rare hereditary disease, characterized by an abnormal blackish coloration of the urine, dark pigmentation of the connective tissue which is due to a deficiency in homogentisate 1, 2-dioxygenase (HGO), a tyrosine catabolizing enzyme. In this study two-years-old girl, five-year-old boy siblings and 11-month-old boy with alkaptonuria were presented. The diagnosis was confirmed by urinary homogentisic acid (HGA) amount. Normal levels of HGA in urine are 2.4 – 12 ng/ml. In these three cases within urine samples, the level of HGA (2.5-dihydroxyphenylacetic acid) showed an increase of 14, 14, 24 fold, respectively. The treatment focuses upon these aspects: correction of the production of HGA and prevention of complications. For reduction of HGA excretion has been suggested treatment with vitamin C and protein restriction have been proposed. Our patients were showed normal growth and development and no major complications of the disease because of consuming a diet with low protein.

Keywords

Kaynakça

  1. Prasad C, Galbraith PA. Sir Archibald Garrod and Alcaptonuria. Clin Genet 2005; 68: 199-203.
  2. Phornphutkul C, Introne WJ, Perry MB, Bernardini I, Murphey MD, Fitzpatrick DL, Anderson PD, Huizing M, Anikster Y, Gerber LH, Gahl WA. Natural history of alkaptonuria. N Engl J Med 2002; 347: 2111-21.
  3. Lorenzini S, Mannoni A, Selvi E. Alcaptonuria. N Engl J Med 2003; 348:1408.
  4. Keller JM, Nercessian OA, Jaffe IA. New developments in ochronosis: review of the literature. Rheumatol Int 2005; 25: 81-5.
  5. Uyguner O, Goicoechea de Jorge E, Cefle A, Baykal T, Kayserili H, Cefle K, Demirkol M, Yuksel-Apak M, Rodriguez de Córdoba S, Wollnik B. Molecular analyses of the HGO gene mutations in Turkish alkaptonuria patients suggest that the R58fs mutation originated from central Asia and was spread throughout Europe and Anatolia by human migrations. J Inherit Metab Dis 2003; 26: 17-23.
  6. Verma SB. Early detection of alkaptonuria. Indian J Dermatol Venereol Leprol 2005; 71: 189-91.
  7. Jeucken YM, Visser G, Jaarsma AS, van Spronsen FJ. A child with dark discoloration of urine. Ned Tijdschr Geneeskd 1999;143: 1641-3.
  8. Morava E, Kosztolányi G, Engelke UF, Wevers RA. Reversal of clinical symptoms and radiographic abnormalities with protein restriction and ascorbic acid in alkaptonuria. Ann Clin Biochem 2003; 40: 108-11.

Ayrıntılar

Birincil Dil

İngilizce

Konular

Sağlık Kurumları Yönetimi

Bölüm

Araştırma Makalesi

Yazarlar

Mehtap Fırat

Murat Sancaktar

Yayımlanma Tarihi

11 Haziran 2010

Gönderilme Tarihi

17 Kasım 2009

Kabul Tarihi

-

Yayımlandığı Sayı

Yıl 2010 Cilt: 32 Sayı: 2

Kaynak Göster

AMA
1.Tanzer F, Ayvaz A, Fırat M, Sancaktar M. Three case reports of alkaptonuria, a rare metabolic disease, in two first-degree relatives and one patient. CMJ. 2010;32(2):235-237. https://izlik.org/JA95HL93NX